Searching journal content for bone metabolism (as phrase) in full text.

Displaying results 1-10 of 40
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  1. Research:
    • Alexander Kot,
    • Zhendong A Zhong,
    • Hongliang Zhang,
    • Yu-An Evan Lay,
    • Nancy E Lane,
    • and Wei Yao
    Sex dimorphic regulation of osteoprogenitor progesterone in bone stromal cells J Mol Endocrinol 59 (4) 351-363, doi: 10.1530/JME-17-0076 first published on 4 September 2017
    ...bone metabolism remains unclear. Our research group reported that global PR-knockout (PRKO) mice had increased bone mass – female PRKO mice had increased bone formation and male PRKO mice had decreased bone resorption compared to 1- to 12-month-old wild-type (WT) mice (Yao et al. 2010). Conditional ~~~
  2. Research:
    • Chunyu Wang,
    • Li Tian,
    • Kun Zhang,
    • Yaxi Chen,
    • Xiang Chen,
    • Ying Xie,
    • Qian Zhao,
    • and Xijie Yu
    Interleukin-6 gene knockout antagonizes high-fat-induced trabecular bone loss J Mol Endocrinol 57 (3) 161-170, doi: 10.1530/JME-16-0076 first published on 4 August 2016
    ...bone metabolism through several potential mechanisms. It is well known that both adipocytes and osteoblasts are derived from the same progenitor cells: bone marrow mesenchymal stem Journal of Molecular Endocrinology (2016) 57, 161–170 Correspondence should be addressed to X Yu Email xijieyu@hotmail. ~~~
  3. Research:
    • Kent Wehmeier,
    • Luisa M Onstead-Haas,
    • Norman C W Wong,
    • Arshag D Mooradian,
    • and Michael J Haas
    Pro-inflammatory signaling by 24,25-dihydroxyvitamin D3 in HepG2 cells J Mol Endocrinol 57 (2) 87-96, doi: 10.1530/JME-16-0009 first published on 27 May 2016
    ...bone metabolism . Steroids 66 375 – 380 . ( doi:10.1016/S0039-128X(00)00155-0 ) ↵ Vernia S , Cavanagh-Kyros J , Garcia-Haro L , Sabio G , Barrett T , Jung DY , Kim JK , Xu J , Shulha KP , Garger M et al. 2014 The PPARα-FGF21 hormone axis contributes to metabolic regulation by the hepatic JNK signali ~~~
  4. Research:
    • Lei Yang,
    • Hongzheng Meng,
    • and Maowei Yang
    Autophagy protects osteoblasts from advanced glycation end products-induced apoptosis through intracellular reactive oxygen species J Mol Endocrinol 56 (4) 291-300, doi: 10.1530/JME-15-0267 first published on 22 February 2016
    ...bone metabolism ~~~
  5. Research:
    • Maxime A Tremblay,
    • Raifish E Mendoza-Villarroel,
    • Nicholas M Robert,
    • Francis Bergeron,
    • and Jacques J Tremblay
    KLF6 cooperates with NUR77 and SF1 to activate the human INSL3 promoter in mouse MA-10 leydig cells J Mol Endocrinol 56 (3) 163-173, doi: 10.1530/JME-15-0139 first published on 12 February 2016
    ...bone metabolism in adults. Despite its essential roles in male reproductive and bone health, very little is known regarding its transcriptional regulation in Leydig cells. To date, few transcription factors have been shown to activate INSL3 promoter activity: the nuclear receptors AR, NUR77, COUP-TF ~~~
  6. Research:
    • Yingyu Feng,
    • Lei Su,
    • Xing Zhong,
    • Wei Guohong,
    • Haipeng Xiao,
    • Yanbing Li,
    • and Lingling Xiu
    Exendin-4 promotes proliferation and differentiation of MC3T3-E1 osteoblasts by MAPKs activation J Mol Endocrinol 56 (3) 189-199, doi: 10.1530/JME-15-0264 first published on 8 December 2015
    ...bone metabolism in aged ovariectomized rats and insulinresistant models. However, whether GLP1 has a direct effect on the proliferation and differentiation of osteoblasts or any cellular mechanism for this potential role is unknown. We examined the effects of the GLP1 receptor agonist exendin-4 on t ~~~
  7. Thematic Review:
    • Evan Simpson and
    • Richard J Santen
    Celebrating 75 years of oestradiol J Mol Endocrinol 55 (3) T1-T20, doi: 10.1530/JME-15-0128 first published on 5 October 2015
    ...bone metabolism Historically, it was assumed that bone growth and function were regulated by oestrogen in women and by androgens in men. However, studies of normal men, those with aromatase deficiency and one individual with a mutation in the ERa, overturned this concept and led to the conclusion th ~~~
  8. Review:
    • Tadeusz Niedźwiedzki and
    • Joanna Filipowska
    Bone remodeling in the context of cellular and systemic regulation: the role of osteocytes and the nervous system J Mol Endocrinol 55 (2) R23-R36, doi: 10.1530/JME-15-0067 first published on 25 August 2015
    ...bone metabolism (SanchezFernandez et al. 2008), the superior role of osteocytes entrapped inside small lacunae of a hard bone matrix in this process was mostly neglected. Despite being the largest fraction of bone cells (w95% of all cells present in the adult skeleton), for long osteocytes were beli ~~~
  9. Research:
    • M Rodríguez-Sanz,
    • N García-Giralt,
    • D Prieto-Alhambra,
    • S Servitja,
    • S Balcells,
    • R Pecorelli,
    • A Díez-Pérez,
    • D Grinberg,
    • I Tusquets,
    • and X Nogués
    CYP11A1 expression in bone is associated with aromatase inhibitor-related bone loss J Mol Endocrinol 55 (1) 69-79, doi: 10.1530/JME-15-0079 first published on 24 June 2015
    ...bone metabolism regulation, offering a new vision of the steroidogenic ability of this tissue and new understanding of AI-induced bone loss.a significant bone loss at lumbar spine (LS) and/or femoral neck (FN) after 2 years on AI-treatment. In contrast, up to one-third (19.6% LS, 38.6% FN) showed no ~~~
  10. Research:
    • Raifish E Mendoza-Villarroel,
    • Mickaël Di-Luoffo,
    • Etienne Camiré,
    • Xavier C Giner,
    • Catherine Brousseau,
    • and Jacques J Tremblay
    The INSL3 gene is a direct target for the orphan nuclear receptor, COUP-TFII, in Leydig cells J Mol Endocrinol 53 (1) 43-55, doi: 10.1530/JME-13-0290 first published on 29 April 2014
    ...bone metabolism in adults. Despite its importance, little is known about the molecular mechanisms controlling INSL3 expression. Reduced Insl3mRNA levels were reported in the testis of mice deficient for chicken ovalbumin upstream promoter-transcription factor II (COUP-TFII), an orphan nuclear recept ~~~
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