Searching journal content for stem cells (as phrase) in full text.

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  1. ...stem cells ~~~
  2. ...stem cells into several cell 313 types including adipocytes (Narvaez, et al. 2013; Valle, et al. 2016). However, there is a gap in 314 our understanding of the consequences of maternal VDD during pregnancy on adipose tissue 315 development and related parameters of metabolic health in offspring. Our ~~~
  3. ...stem cells ~~~
  4. ...stem cells (MSCs). Following matrix deposition and mineralisation, osteoblasts either remain on the surface of the bone as inactive lining cells; undergo apoptosis or become entombed by their secreted matrix and differentiate into osteocytes. Osteocytes reside within the mineralised bone matrix and ~~~
  5. ...stem cells (MSC, precursors for both osteoblasts and adipocytes) lineage commitment toward increased adipogenesis and suppressed osteoblastogenesis. In turn, increased adipogenesis propagates obesity and synthesis of pro-inflammatory cytokines which promote osteoporosis and maintain LGCI in a ‘vicio ~~~
  6. ...stem cells (Berger et al. 1998). Mice lacking the MR developed relatively normally to birth but during the first week postnatally developed symptoms of pseudohypoaldosteronism, losing weight with a failure to survive and eventually died by day 10. MR-null mice displayed hyperkalaemia, hyponatraemia ~~~
  7. ...stem cells are slowly but gradually uncovered, their 23 regulation largely remains virgin territory. Since postnatal stem cells in general reiterate embryonic 24 developmental pathways, attention is first being given to regulatory networks involved in pituitary 25 embryogenesis. Here, we give an ove ~~~
  8. ...stem cells ~~~
  9. ...stem cells ageing: targeting the “purinome” to promote osteogenic dfferentiation and bone repair. Journal of Cellular Physiology 231 1852–1861. (doi:10.1002/jcp.25303) Orriss IR 2015 The role of purinergic signalling in the musculoskeletal system. Autonomic Neuroscience 191 124–134. (doi:10.1016/j. ~~~
  10. ...stem cells (Yagi  et  al. 2013). Specifically, it appears that curcumin inhibits the generation of ROS via alteration of the PI3K/AKT/ mTOR signaling pathway and subsequent inhibition of FOXO1 translocation to the nucleus (Han et al. 2012). In endothelial cells, curcumin protects from oxidative inju ~~~
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